# Hair loss

Category: Conditions

Also known as: alopecia, androgenic alopecia, pattern baldness

Pattern hair loss is best understood not as a genetic or androgen-driven condition, but as a bioenergetic disorder rooted in systemic metabolic stress and local tissue degeneration. The mainstream model, which blames dihydrotestosterone (DHT) for follicular miniaturization, is…

11 passages · 3 authors · 2016–2024 · Most-cited: [Danny Roddy](https://bioenergeticoracle.com/md/voices/danny-roddy/index.md)

Canonical page: https://bioenergeticoracle.com/concepts/hair-loss

## Synthesis

**Pattern hair loss** is best understood not as a genetic or androgen-driven condition, but as a *bioenergetic disorder* rooted in systemic metabolic stress and local tissue degeneration. [Source 1, 2, 11] The mainstream model, which blames **dihydrotestosterone (DHT)** for follicular miniaturization, is a biological paradox because androgens are known to convert vellus hairs into terminal hairs elsewhere on the body. [Source 2, 9] Danny Roddy has argued that the dominance of this theory is an artifact of pharmaceutical marketing, specifically Merck’s development of finasteride, which effectively terminated alternative research pathways. [Source 4] Even James B. Hamilton, the originator of the androgen hypothesis in 1942, acknowledged that the quantities of androgens required for baldness were likely less than those secreted by normal young adult males. [Source 4]

The core pathology involves a shift away from oxidative metabolism, leading to **hypoxia** and a cascade of inflammatory and fibrotic changes in the scalp. [Source 1] Roddy highlights that mast cells are found in significantly higher numbers in balding vertex scalp compared to non-balding occipital scalp, where they degranulate and contribute to *parafollicular fibrosis*, the dissolution of subcutaneous scalp fat, and the accumulation of mucopolysaccharides. [Source 1] This chronic inflammation damages the hair follicle, causing it to enter a prolonged telogen phase, which Roddy reframes not as a resting phase but as a *pre-regenerative phase* following injury. [Source 1] The resulting **oxidative stress** and mitochondrial dysfunction directly enhance hair cell death, creating a self-perpetuating cycle of tissue degeneration. [Source 6]

Ray Peat and Georgi Dinkov emphasize that the hormonal profile of the balding scalp is dominated by stress hormones rather than androgens. [Source 7] Dinkov notes that balding individuals tend to have high levels of **estrogen** and **prolactin** in the scalp, both of which are known to destroy the follicle. [Source 7] Peat observed that topical **DHEA** and copper solutions could slowly regenerate hair, while Dinkov points to a study where administering supraphysiological testosterone to women actually regrew hair, directly contradicting the androgen hypothesis. [Source 7, 8] Roddy synthesizes this by explaining that when thyroid function is suppressed, the gonads produce less DHT, and the body compensates by ramping up adrenal output of DHEA as a protective mechanism against **cortisol**. [Source 3, 9] The elevated DHEA found in the balding scalp is thus a marker of *hyperadrenalism* and systemic stress, not the cause of the hair loss itself. [Source 9, 10]

This systemic energy deficiency also manifests in the brain, which Roddy proposes is intrinsically linked to scalp hair patterning. [Source 5] He cites evidence that aberrant scalp hair directional patterning is associated with problems in early brain development, and that conditions of **cerebral atrophy**, such as Cushing’s disease with its hypercortisolism, are related to androgenic alopecia. [Source 5] The brain’s reliance on sugar means that insulin resistance and the shift to free fatty acid oxidation during stress have a devastating effect on the cerebral cortex, mirroring the metabolic failure occurring in the scalp. [Source 5] Roddy suggests that the high prevalence of digestive issues in those losing hair further supports a picture of systemic dysregulation. [Source 10]

Addressing hair loss therefore requires a multi-faceted anti-fibrotic and pro-metabolic strategy rather than a single targeted agent. [Source 6] Roddy points to substances like **apigenin** and **naringenin**, which downregulate transforming growth factor beta-1 (TGF-β1) and exhibit anti-fibrogenic effects, as well as foundational interventions like vitamin D, calcium, and aspirin to lower parathyroid hormone (PTH), prolactin, and the pro-fibrotic hormone **aldosterone**. [Source 3, 6] Peat’s framework suggests that restoring efficient oxidative metabolism through thyroid support and a diet low in polyunsaturated fats addresses the root cause, allowing the follicle to escape the chronic inflammatory state that forces it into a perpetual cycle of damage and failed regeneration. [Source 1, 6]

## People also ask

### What is the bioenergetic explanation for pattern hair loss?

The entry describes pattern hair loss as a bioenergetic disorder where a shift away from oxidative metabolism causes hypoxia, chronic inflammation, and fibrosis in the scalp, damaging follicles and trapping them in a failed regenerative cycle.

### Why does the entry reject the idea that DHT causes baldness?

It argues the DHT theory is a biological paradox because androgens typically convert vellus hairs to terminal hairs elsewhere, and notes that high estrogen and prolactin in the balding scalp, along with hair regrowth from testosterone in women, contradict the androgen model.

### How does systemic stress connect the brain to hair loss according to this view?

Roddy proposed that the brain’s reliance on sugar means stress-induced insulin resistance and free fatty acid oxidation devastate the cerebral cortex, mirroring the metabolic failure in the scalp, with conditions like hypercortisolism linking cerebral atrophy to androgenic alopecia.

## Related concepts

- [Bohr effect](https://bioenergeticoracle.com/md/concepts/bohr-effect/index.md)
- [Dopamine](https://bioenergeticoracle.com/md/concepts/dopamine/index.md)
- [Hypothyroidism](https://bioenergeticoracle.com/md/concepts/hypothyroidism/index.md)
- [Stress](https://bioenergeticoracle.com/md/concepts/stress/index.md)
- [Thermogenesis](https://bioenergeticoracle.com/md/concepts/thermogenesis/index.md)
- [Vitamin D](https://bioenergeticoracle.com/md/concepts/vitamin-d/index.md)

## Cited passages

Passage numbers match the `[Source N]` markers in the synthesis above.

### Source 1 — Explaining The "Horseshoe" Shape of Male Pattern Baldness

Danny Roddy · Video Transcript · Aug 21, 2016 · https://www.youtube.com/watch?v=0_6ipN3_xb8

> So we hypothesize that this difference in pattern of prostaglandin desynthase expression may constitute a developmental pattern inherent to normal as well as alopecia scalp skin, thus defining a field vulnerable to acquired hair loss. These data indicate that the scalp is spatially programmed via mass cell prostaglandin desynthase distribution in a manner reminiscent of pattern seen in androgenic alopecia. Currently, the main successful treatment options for androgenic alopecia are finasteride and androgen-based systemic therapy with numerous side effects. In a prior study of male pattern alopecia Increased numbers of mast cells have been seen in the balding vertices compared to non-balding occipital scalp. And in fact, this pattern was also observed in five control subjects studied, though there were greater numbers of mast cells in patients with alopecia. In the 1990s, mast cells were found to be actively degranulating in inflammatory infiltrates. of the scalp with male pattern alopecia and this was proposed to contribute to parafollicular fibrosis. So these inflammatory things are changing the scalp and so the typical fibrosis, the edema of the hair follicle, there's also accumulation of mucopolysaccharides, thinner scalp skin, and the dissolution of the subcutaneous scalp fat. So these are all things that are changing the actual scalp because of the recurrent inflammation. And often people say telogen is the resting phase of the hair follicle. And the prominent main feature of so-called male pattern baldness is a higher proportion of telogen hairs to antigen hairs. And that causes the lack of hair growth, obviously. And one paper described telogen is not as a resting phase but a pre-regenerative phase after the hair follicle being damaged. And so that would actually, I think, perfectly fit with the evidence that the scalp is enduring chronic inflammation and it's changing the way the hair grows and it's inhibiting it. So that is my explanation. It goes so much deeper than that. The hair follicle produces its own growth inhibiting hormones like estrogen, cortisol, prolactin, as well as the person producing their systemic hormones. So it gets incredibly complicated, but I want to kind of stop right there. And I think anybody watching might get a general idea of what's suggestive of causing that problem. So thank you guys so much for watching. I sincerely appreciate it.

### Source 2 — Bioenergetic Basics #11: Baldness is Not Genetic (And is Definitely Not Caused by Androgens)

Danny Roddy · Video Transcript · Apr 18, 2024 · https://www.youtube.com/watch?v=8_CT5NSC7RM

> We report 20 pre-pubertal children with androgenic alopecia, 12 girls and 8 boys, age range 6 to 10, observed over the last 4 years. Boldness and polycystic ovarian syndrome, extremely common. So number five, we have pattern boldness post-menopause. So pattern hair loss in women is commoner than described, particularly after menopause. In the absence of other signs of virilization, male pattern hair loss would therefore appear to be a poor indicator of gross abnormality of androgen metabolism. And that was in 1988. Number six, we have the classic androgen paradox. And they say, based on these findings, DHD is held responsible for miniaturization of hair follicles and androgenic alopecia. However, as DHEA is a more potent form of testosterone and androgens are expected to convert hair follicles from vellus to terminal, not the other way around, this interference gives rise to a paradox. In a review, Randall says, how one type of circulating hormone has such contrary effects on a single tissue depending on its body site is not clear. This biological paradox alone makes androgen action in hair follicles very interesting. And the last thing that's being covered up by my graphic here is changes with season. And so this is from Aging Hair again in 2010. And they say such a marked seasonal effect is quite remarkable. Nevertheless, the effect has a major significance as any new drug or treatment should be studied for at least one year to separate any effect from normal seasonal variations. So that's it for this first part on hair loss, and we'll get into a second part of what I think is actually happening next two weeks. And first, we need to talk about the course. So I put together a bio energetic basics course, and this is based on talking to many, many, many people over the last ten years. And so I know I noticed that similar questions would pop up over and over and over again. And some of those questions, there was a lot to take in at the same time. Like for example, if a person is going to investigate taking thyroid, there are a lot of little things to know that if a person just didn't pay attention, they could cause some damage.

### Source 3 — #50: DHT for Hair Growth? | New Ray Peat Interviews | Keto and Mitochondria | Childhood Trauma with Georgi Dinkov

Georgi Dinkov · Interview · Feb 20, 2021 · https://open.spotify.com/episode/3G7mHN8kzZb9bqFjuma0iq

> **Danny Roddy:** I feel like aldosterone should be included in that conversation because that's actually one of the more well-studied aspects of so-called androgenic alopecia that they've measured in men and women. And that's associated with the hypertension or causes the hypertension.
>
> **Georgi Dinkov:** And then, didn't Ray- It's a pro-fibrotic hormone. And in some countries, the spironolactone drug, which is the main aldosterone antagonist in clinical use, it's actually approved for hair loss treatment.
>
> **Danny Roddy:** And then that kind of segues into, Ray is always talking about vitamin D and calcium for hair loss. Well, those lower PTH and prolactin, but they also, I think by extension, because they lower PTH, lower aldosterone as well. But just to go over this, to maybe sell somebody on the idea. So if baldness is caused by lower thyroid function or high stress, the thyroid function is suppressed from many different variables, including the accumulation of PUFA. Testicles don't work very well when thyroid is suppressed. You're going to increase the hypothalamus pituitary and adrenals, and the dihydroepiandrosterone, the DHEA, will be metabolized peripherally into androgens. And so if they do a biopsy of skin on the scalp or something, that might explain why there's a couple of papers about higher levels of androgen in skin. Yeah.

### Source 4 — Bioenergetic Basics #11: Baldness is Not Genetic (And is Definitely Not Caused by Androgens)

Danny Roddy · Video Transcript · Apr 18, 2024 · https://www.youtube.com/watch?v=8_CT5NSC7RM

> Therefore, one of the prime challenges of modern hair research is a more profound understanding of the molecular controls of hair follicle cycling. Common diseases such as alopecia areata, telogen effluvium, and androgenic alopecia until then will remain unsolved medical problems. So why is it unsolved medical problem? We don't have time to get into it too much, but I think Merck ruined pattern baldness research. I don't think it would be as bad as it is now if Merck hadn't gotten involved. And that is a long story. And it's in this article from 1992 called Keeping the Pipeline Filled at Merck. I'll just read a little bit here. They say, every company has its legends. At Merck and Company, the latest is about ProScar, the new drug that shrinks in large prostates and is awaiting government approval. It involves a remote Caribbean mountain village with a population of male pseudo-hermaphrodites and an obscure medical paper about them. P. Roy Vegalos, Merck's chairman, seems never to tire of telling the story, and anyone inquiring about ProScar's prospects as the company's new billion-dollar megadrug is sure to hear about it from the Merck executives as well. And no wonder, since the ProScar legend says a lot about survival and the bare-knuckle competitiveness of the drug industry these days, illustrating how basic research and unrelenting patience must be combined to bring a drug to market. Aside from terminating effective hair loss research with the invention of finasteride and therefore supporting in a lot of people's minds that hair loss was in fact from androgens. I don't think finasteride works by inhibiting androgens. I think it has a different mechanism, but there's really no reason to not trust Merck. They're a good company aside from ruining pattern baldness research. Okay, so let's talk about the second thing that I think is a huge problem is the idea that masculinity was the real problem with pattern baldness. So here's a paper from Neves in 2014, and they say current treatments are based on pathophysiological models, which have not significantly gone beyond Aristotle's observation that eunuchs do not go bald. This is partly because our knowledge of the pathogenesis is still meager and offers few rational targets for therapy.

### Source 5 — Brain Atrophy in Androgenic Alopecia? (My New Theory)

Danny Roddy · Video Transcript · Apr 8, 2019 · https://www.youtube.com/watch?v=956IF0oDfZY

> And those all... are conditions of cerebral atrophy or problems with the different areas of the cerebral cortex. And that can also manifest as male pattern or androgenic alopecia or whatever you want to call it, pattern baldness. And so that was very interesting to me. And again, this hasn't been sussed out in the research to my knowledge. And so again, I'm putting forth kind of a hypothesis that needs to be tested in some capacity. And just the effects of stress on the prefrontal cortex and the parietal cortex and things like that is just not controversial. And that is demonstrated really well in Cushing's disease, so hypercorticalism. And cortisol is something that has come back in males and females as being something that not only has a devastating effect on hair growth, but is related to so-called androgenic alopecia. In this paper in 2000, they say hypercorticolismic state manifested in patients with Cushing's disease promotes the premature development of cerebral atrophy. And again, if the brain can't get sugar or there's insulin resistance, it's going to have a devastating effect on the brain's metabolism and the storage of glycogen. And that... uh i don't have the paper in front of me but there was a a paper that talked about how the brain like albert st georgia has a great quote where he talks about how nature builds on its structures on top of old structures and the cerebral cortex is the newest structure on top of the older more like primitive structures And it was the newest structures that were most devastated by things like stress and inflammation and aging and things like that. So again, that would just tie in perfectly with the idea of insulin resistance being more likely as a person gets older, relying on free fatty acids instead of sugar, being less able to store glycogen efficiently and just all the general generalized things that happen with aging and stress as they accumulate. So all that to get to this hierarchical approach of pattern baldness. This is just me spitballing. I don't think this is not a religious text. It can be changed and made better. I'm just throwing things out there. But just from my experience of talking to people, things I've experienced myself, I threw this together.

### Source 6 — Reversing Fibrosis in Male Pattern Baldness

Danny Roddy · Video Transcript · Nov 12, 2016 · https://www.youtube.com/watch?v=CS9ygUEQ3E8

> And that's, I think there's some suggestions in the research that are actually jive with that point of view. So this one paper from 1985 said androgenic alopecia is probably a series of diseases with a common end pathway. And then another paper in 2014 said it is unlikely that a single targeted agent will be sufficient for treating androgenic alopecia and therefore it would be a challenge to obtain compounds with multiple activities and so to tag along on that there was a book on fibrosis and just the the pathology of fibrosis and they thought that an anti-fibrotic therapy would include things that will one removing causative agents agents for cytoprotection or cell protection anti-inflammatory agents and anti-transforming growth factor beta-1 agents or tgfb1 and so considering we already know that progesterone is helpful you need enough thyroid to produce normal amounts of progesterone in both men and women And so I could talk about milk. I could talk about cheese, their calcium content and the protein's ability to increase IGF-1 and those foods having IGF-1 in them already. Fructose, strong coffee, a diet low in the polyunsaturated fats, vitamin D, vitamin K2, antibiotics. But I'm not going to talk about those things. I'm going to talk about some other things. uh things that i talk about less often so uh the first one i wanted to talk about was apigenin and i'm not really sure if i'm saying that right i've never heard anybody say it but apigenin is a chemical found in guavas and mushrooms and it is a really interesting chemical it has a direct relation to hair loss so they say Taken together, these findings suggest that apigenin, which is known to have an antioxidant, anti-inflammatory, and anti-tumor property, stimulates hair growth through the downregulation of transforming growth factor beta-1 gene. That was done in 2009. And apigenin is also an aromatase inhibitor. So they say several naturally occurring synthetic flavones were found to inhibit aromatization. These flavones include apigenin. Back to the first apigenin reference. It's a powerful anti-inflammatory. So they say the study suggests that the modulation of COX-2 and inducible nitric oxide synthase by apigenin and related flavonoids may be important in the prevention of carcinogenesis and inflammation.

### Source 7 — #47: The Power Elite  |  Vaccines  |  Hair Loss  |  UFOs  |  Surviving The Great Reset with Ray Peat, PhD

Ray Peat (with Georgi Dinkov) · Interview · Jan 23, 2021

> ## Hair Loss and Hormones
>
> **Danny Roddy:** Austin asks about vellus hairs turning to terminal hairs. Babies have thin hair that turns thick; balding people have the reverse. Is it possible to turn vellus hairs back to terminal hairs?
>
> **Ray Peat:** I've experimented several times with using DHEA in oil on the sides of my eyebrows that have gradually gone through aging and disappearance. It took about six months to create new, dark, normal hairs. Using a topical copper solution, the very next day I could see the colored hair beginning at the base. In the case of lost hairs, it was a very slow process.
>
> **Georgi Dinkov:** I think there's a difference between the hair type a baby has and a balding person has. Babies have low androgens and high pregnenolone. Balding people tend to have high levels of estrogen and prolactin in the scalp. Both prolactin and estrogen are known to destroy the follicle.
>
> **Ray Peat:** There have been interesting studies on dogs. Certain dogs became completely bald when their owners started supplementing estrogen on their skin.
>
> **Georgi Dinkov:** Isn't a good indication that estrogen and prolactin are involved in hair loss the fact that most menopausal women start having serious problems with hair thinning?

### Source 8 — #50: DHT for Hair Growth? | New Ray Peat Interviews | Keto and Mitochondria | Childhood Trauma with Georgi Dinkov

Georgi Dinkov · Interview · Feb 20, 2021 · https://open.spotify.com/episode/3G7mHN8kzZb9bqFjuma0iq

> **Georgi Dinkov:** I don't know. Yes, yes, it was pellets. But it's the same thing. I mean, basically, like, here we go. If the androgens were behind hair loss, right, you should have. And actually, if you see, if you look at the warnings on testosterone therapy for both men and women, actually, they say one of the side effects, one of the common side effects is hair loss, right? But this study found the exact opposite. You know, if if testosterone is the cause of the... And actually, they actually administered, you know, slightly higher than physiological amounts to women of the testosterone. And it's still basically like, I think 60% or something experienced hair regrowth. So again, you know, I don't know. So the androgenic alopecia is not only a condition for men. It also happens in women. And in fact, basically, it's one of the science... slash symptoms of PCOS, polycystic ovarian syndrome, right? And they're saying, oh, we know this is, because this syndrome is caused by androgens, therefore, androgens must be also basically, and because these people also experience hair loss, ergo, androgens are causing the hair loss in women too, the androgenic alopecia in women too. But it's not. It's basically, look, it's just the administration of super physiological testosterone doses to women regrow their hair.

### Source 9 — The Misunderstood Role of DHT in Male Pattern Baldness

Danny Roddy · Video Transcript · Oct 27, 2016 · https://www.youtube.com/watch?v=5Greaw5MK9k

> And then two just points for androgens growing hair is that women with androgenic alopecia, one study found that testosterone was anabolic for hair growth. So replacing testosterone in women with androgenic alopecia regrew hair. And then similarly in 1965, topical administration of testosterone regrew hair in older men. So two things to keep in mind. Number two, what's the relationship of testosterone and DHT to baldness? Like, is there any relationship? And I covered this a little bit in Organizing the Panic, but I just wanted to go over it here too. So, I'll read a couple studies. One's from 1979. They say, we conclude that bald men are no more masculine than those with good scalp hair growth if masculinity is defined in terms of an organ response to androgenic stimulation. One from 2014, they say increased serum concentrations of DHT were not correlated with the advance of alopecia. Based on these results of our study and others, the most important factors would appear to be genetically determined sensitivity of the follicles to DHT and their different reactions to androgen concentration. And of course, I disagree with that last part and I think that's kind of an easy wiggle way out of the wrong theory. And finally, it is well known that the androgenic alopecia patient's blood concentration level of male hormones are not different from those people without alopecia. And also, I'm going to flash multiple studies from female androgenic alopecia. And it's a little more free thinking over in that realm. And they talk about hypothyroidism causing androgenic alopecia in women. But still, they're kind of mystified by the idea of prolactin and TSH being related to androgenic alopecia. And also the two theories don't line up at all. So if you want to have androgenic alopecia as a theory of baldness, it doesn't even come close to explaining androgenic alopecia and women. So the theories are discordant and they don't make any sense. And for women, different factors are involved. But for men, it's only this weird DHT and unknown mechanism in genes and things like that.

### Source 10 — Thermo Diet Podcast Episode 36 - Danny Roddy

Danny Roddy · Interview · Jun 14, 2020 · https://www.youtube.com/watch?v=p-S0wfDbWeI

> **Thermo Diet:** Yeah, it's quite fascinating. I was blown away whenever I thought about it. And it wasn't too far after I was reading your LSD article too. So it kind of just clicked whenever I was reading it. But so there is a clear connection between serotonin and hair loss. Can you kind of walk us through how that works?
>
> **Danny Roddy:** Yeah, so I don't think there's any paper that studied it. But I think, again, if cortisol is elevated in patterned boldness, and I think there is, again, this area is completely understudied. And so we're kind of like grasping at straws here. I think my main position would be like, hey, look, the genetic androgen hypothesis we've had. how many decades of this theory and it's produced drugs like finasteride and minoxidil and to a lesser extent nizrol this thing is like a total failure in my opinion and there's some like ideal ideological commitment to this point of view that people just want to um maybe they're taking a drug and you know they're crossing their fingers and they're really hoping it works or whatever it's like their only chance But I think it kind of clouds the judgment of thinking about it in a new way or like tackling the problem from just like a different angle. And so that's what I tried to do in my, I wrote a book in like 2013 and I tried to go through how the theory was concretized and like how it came to be. And it's like really flimsy, you know? And so, yeah. You were asking about serotonin and cortisol and prolactin. So I guess if you find these intermittent papers that talk about elevated prolactin in so-called female androgenic alopecia and then male androgenic alopecia so-called, and then you find prolactin, there's a few papers that talk about that being elevated. I did a live stream on the hyperadrenalism of patterned baldness. So this is something that's been kind of well studied. Not well studied, but there are more than one or two papers on it. So young men tend to have like very high DHEA levels and apparently DHEA rises to meet cortisol during a stressful event. And this is a protective thing that DHEA does, but over a prolonged period of time, like the certain layers of your adrenal glands deteriorate and then you're left with just cortisol and not enough DHEA.

### Source 11 — Ep. 99 Interview Danny Roddy: The Wisdom of Ray Peat, and How Elevation Will Help You Live Longer

Danny Roddy · Interview · Nov 24, 2021 · https://www.youtube.com/watch?v=W7F-rIgoHd8

> **Lucas:** Hey, everyone. Welcome to the Quacks Podcast. So today I have on Danny Roddy, the myth, the legend. Danny is a Ray Peat guy, if you've never heard of him. He interviews Ray Peat and Georgi Dinkov quite often on his own podcast. It's a great show. So this is going to be a bit of a Ray Peat subject matter episode, that's for sure. I first talked to Danny back in 2015 or so when he was getting his Patreon going and he was starting to do coaching. Now at the time, Danny was well known for his book, which was titled Hair Like a Fox, A Bioenergetic View of Pattern Hair Loss. I'd read the book at the time and it was an alternative explanation of balding. I found it very interesting. The mainstream explanation is genetics and androgens, of course. But Danny came at it from this Ray Peat perspective, which was really cool. It was really refreshing, and it got a lot of people's imaginations fired up. I still think his book is a great introduction to some of Ray's concepts and how they can be applied to physical symptoms. So if you are interested in reading it, I will have it in the show notes. Now, Danny, he's got a really interesting backstory, which we will get into. He was actually in a band called Dakota that was signed by Island Records back in the early 2000s. I think he was the bassist, but there are music videos of him like playing music and stuff. It's really cool. You can still find it. Anyways, all that is to say, you know, I feel very privileged that he came on the podcast and the knowledge he shares of his experience with Peat and the principals. It's just awesome. So enjoy the show. All right, everybody. Welcome to the Quacks podcast. It is my pleasure to introduce Danny Roddy onto the show. It feels like it's been something that should have happened a long time ago, but I'm glad that it's happening now. Danny, welcome to the show.
>
> **Danny Roddy:** Better late than never. Thanks for having me on, Lucas.

_Generated 2026-07-20 from the Bioenergetic Oracle corpus._